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D increases permeability of small blood vessels and smooth muscle contraction. In macrophages, eosinophiles, and neutrophils anaphylatoxins can induce oxidative burst, basophiles, and mast cells release histamine, and C3a can enhance the effect of other proinflammatory cytokines such as TNF, IL-6, and SDF-1. While the mechanism for the many reactions precipitated by complement anaphylatoxins has n
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Known to be "promiscuous" [109] with multiple binding partners. This promiscuity most likely is due to the multiple surfaces presented by the IDPs or the low energy requirement associated with their role in mineralization. Thus, it is not surprising that DMP1 can interact with cell membrane receptors (e.g. v3 integrin [122] to regulate signaling [123], FGF23 expression [66], bind to HA crystals so
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Hemoglobin subunit alpha (Hemoglobin alpha chain) (Alpha-globin) Keratin complex 1, acidic, gene 10 Kpnb1 protein b Lactate dehydrogenase 2, B chain Murinoglobulin-1 presursor (MuG1) Myosin heavy chain IIB Peroxiredoxin 1 Prothrombin precursor (Ec 3.4.21.5) (Coagulation factor II) Pulmonary surfactant associated protein A precursor (SP-A) (PSP-A) (PSAP) Rho GDP dissociation inhibitor (GDI) alpha S
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E inflammatory response and tissue remodeling in tendon healing and also it revealed that; although the severe inflammatory reaction has been developed in response to the collagen implant, but this immune response was due to the remodeling effect of the collagen implant, not its rejection. It has been postulated that inflammation has a major role in tendon healing and if immune response does not p
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Their growth ad matrix crack population). Raine's syndrome, a rare condition with an incidence of 1/1,000,000, is due to a mutation in FAM20C the Golgi protein kinase that phosphorylates most secreted phosphoproteins including SIBLING proteins [160,161]. The osteosclerosis characteristic of this skeletal dysplasia provides clinical support for the importance of controlled phosphorylation of IDPs f